Prevention and treatment of coccidiosis in poultry
An Innovative SAFE & NATURAL Solution
VemoHerb® deCox’s proprietary blend of Artemisia spp. extract supports the prevention and treatment of coccidiosis hence reducing the economic impacts through enhanced feed conversion ratios and reduced mortality.
VemoHerb® deCox is formulated with a blend of herbs with a special emphasis on artemisinin, derived from Artemisia species for a natural control of coccidiosis. In contrast to many synthetic coccidiostats, this method does not develop drug-resistant Eimeria strains, which means that its effectiveness does not decrease with time. It mitigates gut inflammation, enhances recovery rates, and decreases mortality, making it a suitable option for poultry producers who are on constant lookout for better and safer solutions!
Overuse or misuse of anticoccidial drugs can result in drug-resistant strains of coccidia, making disease control more challenging and often requiring more expensive or alternative treatments.
Coccidiosis compromises the immune system, increasing their vulnerability to secondary infections. This not only complicates disease management but can also further impact flock health and productivity.
Managing coccidiosis comes with substantial costs, including the use of anticoccidial drugs, vaccines, and the labor required to treat infected flocks. These expenses can significantly impact a farmer’s overall profitability.
VemoHerb® deCox can be used for:
VemoHerb® deCox is based on the combination of phytogenic extracts, with the main ingredient of Artemisinin from different Artemisia species.
Product B is based on concentrated plant steroidal saponins, extracted from Yucca and Trigonella species.
Test groups:
Negative control – Chickens without challenge and treatment
Positive control – Chickens were challenged at day 21 but untreated
Diclazuril prevention – Chickens were challenged at day 21 and treated with 0.2 g/Kg of diclazuril (prevention dose) from day 1 to 42.
Diclazuril treatment – Chickens were challenged at day 21 and treated with 0.2 g/Kg of diclazuril (prevention dose) from day 1 to 20 and treated with 0.8 g/Kg of diclazuril (treatment dose) from day 21 to 42.
VemoHerb® deCox prevention – Chickens were challenged at day 21 and treated with 0.2 g/kg of VemoHerb® deCox (prevention dose) from day 1 to 42.
VemoHerb® deCox treatment – Chickens were challenged at day 21 and treated with 0.2 g/kg of VemoHerb® deCox (prevention dose) from day 1 to 20 and treated with 0.8 g/kg of VemoHerb® deCox (treatment dose) from day 21 to 42.
Product B prevention – Chickens were challenged at day 21 and treated with 0.2 g/kg of product B (prevention dose) from day 1 to 42.
Product B treatment – Chickens were challenged at day 21 and treated with 0.2 g/kg of product B (prevention dose) from day 1 to 20 and treated with 0.8 g/kg of Product B (treatment dose) from day 21 to 42.
Among the challenged chickens, the lowest percentage of necrosis and epithelial tissue destruction was observed in the cecum and jejunum of chickens in the VemoHerb® deCox and diclazuril treatment groups.
Among the challenged groups, numerically, the lowest number of oocytes was counted in the feces of chickens fed with diclazuril and VemoHerb® deCox.
Production plant:
South Industrial Zone,
5835 Knezha, Bulgaria
Headquarters:
88 Rozhen Blvd.
1271 Sofia, Bulgaria
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